By Khalid Iqbal, Sangram S. Sisodia, Bengt Winblad
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Additional info for Alzheimer z disease advances in etiology, pathogenesis a therapeutics
This instrument assesses multiple cognitive abilities, including orientation as to time and place, immediate and delayed word recall, naming, verbal repetition, reading, writing, and spatial ability. , 2000). This result supports the view that the earliest cognitive deficits in AD are seen within the domain of episodic memory. 2. , 2001) reported the results from a comprehensive neuropsychological battery administered to a sample of the entire Kungsholmen Project’s cohort. This sample was examined three times during a follow-up period of six years.
Brain, 121, 1601–2. Howieson DB, Dame A, Camicioli R, Sexton G, Payami H, Kaye JA (1997) Cognitive markers preceding Alzheimer’s dementia in the healthy oldest old. J Am Geriatr Soc, 45, 584–9. Jacobs DM, Sano M, Doonaief G, Marder K, Bell KL, Starn Y (1995) Neuropsychological detection and characterization of preclinical Alzheimer’s disease. Neurology, 45, 957–62. Killiany RJ, Gomez-Isla T, Moss M, Kikinis R, Sandor T, Jolesz F, Tanzi R, Jones K, Hyman BT, Albert MS (2000) Use of structural magnetic resonance imaging to predict who will get Alzheimer’s disease.
In the projection of dementia prevalence in the developed world, we used the Canadian prevalence rates since this is the largest population-based study that contains significant numbers of subjects over age 90, the age group that will increase in numbers most rapidly in the next 50 years. What happens to the exponential increase in dementia at advanced ages has been a matter of controversy. Hagnell et al. in 1981, on the basis of data from the Lundby study, suggested that the risk of dementia might decrease after age 85.
Alzheimer z disease advances in etiology, pathogenesis a therapeutics by Khalid Iqbal, Sangram S. Sisodia, Bengt Winblad